RCMI Coordinating Center (RCMI CC) Header Logo

Search Result Details

This page shows the details of why an item matched the keywords from your search.
One or more keywords matched the following items that are connected to Carbone, Michele
Item TypeName
Academic Article Programmed necrosis induced by asbestos in human mesothelial cells causes high-mobility group box 1 protein release and resultant inflammation.
Academic Article Cancer cell secretion of the DAMP protein HMGB1 supports progression in malignant mesothelioma.
Academic Article Continuous exposure to chrysotile asbestos can cause transformation of human mesothelial cells via HMGB1 and TNF-a signaling.
Academic Article HMGB1 and Its Hyperacetylated Isoform are Sensitive and Specific Serum Biomarkers to Detect Asbestos Exposure and to Identify Mesothelioma Patients.
Academic Article HMGB1 targeting by ethyl pyruvate suppresses malignant phenotype of human mesothelioma.
Academic Article Expression of Concern: HMGB1 and Its Hyperacetylated Isoform are Sensitive and Specific Serum Biomarkers to Detect Asbestos Exposure and to Identify Mesothelioma Patients.
Concept HMGB1 Protein
Grant Germline BAP1 Mutations and Malignant Mesothelioma: Mechanisms and Early Detection
Academic Article Asbestos induces mesothelial cell transformation via HMGB1-driven autophagy.
Academic Article Mesothelioma Biomarkers: Discovery in Search of Validation.
Academic Article HMGB1 as a therapeutic target in disease.
Academic Article BAP1 forms a trimer with HMGB1 and HDAC1 that modulates gene ? environment interaction with asbestos.
Academic Article Preventive and therapeutic opportunities: targeting BAP1 and/or HMGB1 pathways to diminish the burden of mesothelioma.
Academic Article HMGB1 released by mesothelial cells drives the development of asbestos-induced mesothelioma.
Search Criteria
  • HMGB1 Protein
RCMI CC is supported by the National Institute on Minority Health and Health Disparities, National Institutes of Health (NIH), through Grant Number U24MD015970. The contents of this site are solely the responsibility of the authors and do not necessarily represent the official views of the NIH

For technical support please contact support